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Duralactin-Treating Arthritis

A NEW APPROACH TO TREATING ARTHRITIS/INFLAMMATION ?

It appears that a new product has been discovered to treat or manage chronic inflammatory conditions including Osteoarthritis and soft tissue injury.

This product is NOT a drug and is NOT a steroid, but believed to be an immuno-nutritional aid. It apparently relieves clinical signs associated with inflammation. It is intended for use as a nutritional aid for the management of chronic inflammatory conditions including musculoskeletal disorders in aging pets.

This product contains Microlactin, (dried milk protein) which has natural anti-inflammatory properties. Microlactin is a patented, milk derived, high protein product from hyperimmunized cows and has the ability to stop the inflammation process earlier.

It has no know side effects, is apparently safe for long-term use and may be used alone to manage inflammation or in conjunction with NSAIDS such as Rimadyl or corticosteroids. It also seems to have no evidence of stomach or bowel irritation. It has shown no toxicity in humans or animals in the studies done.

Each 2.4mg tablet of Duralactin Canine contains 1000mg of Microlactin (dried milk protein), dextrose, stearic acid, vanilla flavouring and magnesium stearate. All ingredients are food grade.

Dosage is as follows :

  • Dogs 0 – 40 pounds : ½ tablet, twice daily

  • Dogs 40 – 80 pounds: 1 tablet twice daily

  • Dogs 81 – 120 pounds: 1 ½ tablets twice daily

Dog Food Recall


written by Robert Hart, Vitality Science

Largest Recall Of Pet Foods In History: The ongoing pet food recall offers a glimpse of what is wrong with entrusting your pet´s nutrition (or your own) to corporations and government regulatory agencies.

Scary Ingredients Approved By The Government: Why is “gluten”, or “rice protein concentrate” even in pet food? Answer, there is a constant search by manufacturers to produce lower cost food that still meets government protein and nutrient requirements. Gluten and rice protein are  not the only cheap and threatening additive approved by the government and added by manufacturers.

Dry Pet Foods are much like breakfast cereals. They are cooked in a mash, then extruded through nozzles at high heat and pressure. This process not only destroys nutrients, it makes much of the rest indigestible. If the ingredients originally had any nutritional value, the extrusion process is sufficient to destroy them. About the best things you can say about dry pet food is that they are “cheap and convenient”!

Feral cats and dogs would have to be starving before they would eat the ingredients found in dry pet foods. If for no other reason, these ingredients do not provide the nutrients they need to sustain life, let alone health.

Wet Pet Foods are made from the most distressed meat, animals with pus bags and tumors. Even though these animals are approved by the USDA for human consumption, some, but not all, manufacturers are reluctant to use them for humans. Even the meats that are not visibly distressed contain growth and fattening hormones, antibiotics, and highly questionable feed. Additionally, among many scary ingredients, pet foods may legally contain euthanized cats and dogs!

If you own a cat or dog, Google “Dog Food Secrets”. I was aware of much of what this book exposes, but it still left my mouth agape.

Pay Me Now, or Pay Me Later: GI and immune diseases are at an all time high. Humans and animals are on multiple medications. What are the common denominators? Fast, nutrition-less foods, chlorinated drinking water, not incorporating bioavailable vitamins, supplements and digestive aids into the daily diet, and lack of exercise.

GI disorders among cats and dogs is epidemic. Conventional medication treatment is archaic, potentially causing further damage to animals already weakened from lack of proper nutrition.

According to The Royal Society of Medicine Great Britain, “Fully 90% of all chronic disease is caused by an unhealthy intestinal system.”

It´s OK to Domesticate Cats and Dogs, but Not Their Diets

The original purpose of domesticating dogs and cats was cheap labor. Cats are used by farmers as mousers, dogs as herders, hunters, and protection.

These working relationships no doubt led to some close relationships. Over time, working animals (or more likely their offspring) were sold or given away to city folk.  Today, over 90 million suburban households own an average of 1.67 cats or dogs, or over 150 million domestic cats and dogs.

Wild or feral animals hunt and eat fresh kill. They also instinctively eat grass and dirt for the micronutrients and soil-based organisms needed to properly digest the meat.

When they kill an herbivore they first consume its´ entrails which usually contain plenty of greens and digestive enzymes needed to break down, digest and eliminate the flesh. The closer pet owners come to replicating the diet of their feral relatives, the healthier and happier their domestic pets will be.

Genetic Predisposition at The Blink of an Eye

Without healthy foods, even healthy animals will decline genetically within several litters.

The best way to assure that your pet lives a long healthy life is to know how well the breeder cared for the mother, and the mother´s mother. Pets are capable of having multiple litters within their lifetime. This is common in feral animals and commercial breeders (mills). Each subsequent generation inherits the weaknesses of the previous generations. Today, many millions of kittens and puppies are predisposed to an unhealthy life.

Resorting or Maintaining Pet Health:
  The good news: Animals that are predisposed to diseases are less likely to develop them when fed a healthy diet. Genetic predispositions can be bred out over several generations of proper feeding.  

It is up to the pet owner to take responsibility for the health and wellbeing of their pets. Like food choices, choose your “professionals” carefully. Veterinarians that recommend a raw food diet combined with supplements and digestive aids- before medications- have your pet´s best interest at heart!

Local bookstores, libraries, and the internet offer a wealth of knowledge regarding proper care and feeding.  The American Holistic Veterinarian  Association website has a national directory.

TEASER COPY: The recent pet food recall offers a glimpse of what is wrong with entrusting your pet´s nutrition to corporations. When it comes to Pet Food, it seems there is no Consumer Advocacy Group looking out for pet owners. You Can Domesticate Cats and Dogs but Not Their Diets.

Dog Food Dilemma

Imagine yourself driving down the highway in your Rolls Royce. You peek down at the gas gauge and see your machine is getting hungry for fuel so you pull up to the service station. There are five pumps marked like so: Premium $1, Regular $.75, Diesel $.80, Natural Gas $.60 and a final pump marked “Leftover Crap” $.10. What do you do? It’s a Rolls for goodness sake! Of course you put in the Premium. What’s the point in buying a $180,000 machine and putting in a fuel that either reduces its performance such as Regular would do or damages the machine (such as the other choices would do)?

Our dogs’ bodies are machines, and the work we train them to do is the performance we expect from their machines. And if we are expecting high levels of performance, then we must give them the right fuel to achieve that.

But choosing the right fuel can be more difficult than you think. Look back at your Rolls Royce. Some choices are obvious, some are not. It doesn’t take a lot of grey matter to realize that the mystery brew “Leftover Crap”, made up of a bit of Regular, a bit of Diesel, a bit of water and a bit of dirt just isn’t going to allow your machine to work right. But your other choices – Diesel and Regular – they aren’t “bad” fuels. They are simply not what your Rolls was designed to take.

What fuel was your dog’s body designed to take? There are lots of clues. Look at his teeth. Fangs, designed to seize and kill prey. His molars are sharp wedges to shear bone and connective tissue, unlike yours which are flat blocks to allow grinding. Look at his intestine: short, to process small amounts of highly concentrated foods (compare that to herbivores which have long digestive tracts to process high volumes of foods low in nutrient concentration). Look at what his wild relatives, feral dogs, wolves and coyotes, are eating. The conclusion is that our dogs are carnivores and their diet consists primarily of the bodies of other animals. That is: muscle, fat, bone, brains, organs, eyes, eggs and the like.

Now look at the label of the dog food you are feeding your carnivore. Rice, wheat, corn, bran, beet pulp, with a little meat or meat by product thrown in. Now, don’t get me wrong: there is nothing wrong with rice or wheat – I eat them all the time. But then, I’m not a carnivore. And there is nothing wrong with diesel … my neighbour’s tractor runs just fine on the stuff. But it’s not a Rolls Royce.

I ask you to start from this basic, self evident proposition: Mother Nature knows best. We have been telling our dogs for a very long time now that in fact we know best. We know that balanced nutrition is important, and we know we can achieve that balance predominantly with the inexpensive grains rather than those expensive meats and fats. Or at least the National Research Council and the pet food industry know. And they in turn have trained our veterinarians on pet nutrition, so now our vets know. But no matter how much we tell this to our dogs, they remain unconvinced.

You see, we animals all consume six key nutrients: water, fat, protein, carbohydrate, vitamins and minerals. The one thing we all have in common is that we should have ample and unrestricted access to water. After that, the agreement breaks down, with each species thriving on different combinations of the other five nutrients than any other species. Let me give you an example. We humans with our moderately long digestive tracts need a moderately high level of fibre in our diets. So for years you’ve been told by doctors and dieticians: eat more grains and veggies. And of course we don’t want our doggies to get colon cancer so we make sure they get a solid 4 or 5 % – or more – of fibre too. But their digestive tracts are unlike ours; they are short and not prone to cancer. Yet we are stuffing more than thrice as much fibre down them than is typically in our own diets! And not only don’t they need it: the presence of all this fibre is actually an irritant to their carnivorous guts. So once again we are back to the initial principle: don’t feed tractor fuel to a Rolls Royce.

I’ve read dozens of pet food labels and they almost always tout the quality of their carbohydrates (rice, wheat, other grains) as a good source of energy with which to fuel activity. But carbohydrates, like proteins, contain only four calories of energy per gram. Where do you think a carnivore is going to naturally look for his energy: grazing on five pounds of cud or stripping one pound of fat from his prey?

But what about that nasty cholesterol and heart disease that we are all afraid of? We don’t want that for our dogs, do we? Well of course not. But again, we cannot blindly assume that everything about our omnivorous bodies is the same for the carnivorous bodies of our canine friends. And indeed there is a significant difference on this very point. Their digestive system is far more adapted to handle fats than ours is, and they do not suffer from these same dietary restrictions (cholesterol and dietary heart disease). Research has shown no ill effects on dogs routinely fed diets as high as 65% fat. (You or I would live to a ripe old age of 6 on such a diet, but dogs thrive on it.) That same research has shown that dogs fed fat-rich diets enjoy more endurance than dogs fed low-fat diets such as the pet food companies make.

And what about all those valuable carbohydrates (which makes up 50 – 60% of pet food, or more)? Research has shown little need for them in our dogs’ diets. They are pumped into dog food for one reason: food energy (i.e. – calories). (Okay, maybe one more reason: they’re cheap, waaaay cheaper than fat). But as already discussed, fat is a dog’s preferred energy source. In nature, wild canines eat relatively little material from plants (the source of carbohydrates) and speculation is that most of that small amount they do eat (berries, stomach contents of their prey) is consumed not so much for the carbohydrate and those 4 calories per gram but instead for the vitamins which those plants offer.

Another key ingredient in diet is protein. Unlike either fats or carbohydrates, which are primarily fuel sources to the body, protein is a structural component and more. While it can be used as a mere fuel to produce 4 calories of energy per gram, it’s primary roles are instead to: make muscle, blood, bone and organ tissue; create enzymes which facilitate the myriad of chemical reactions needed to sustain life; comprise hormones which regulate body functions; and develop the immune system that protects the dog’s body from disease and infection.

Protein is actually not a single “thing” but a bunch of related “things” more properly called amino acids. There are 23 amino acids, and like us our dogs can make some but not all the amino acids needed to perform the various functions of proteins. In fact the dog cannot make 10 of those amino acids but must instead obtain them from the food he eats. Those 10 are called essential amino acids. (Human adults have only eight essential amino acids and children have nine.) And essential amino acids can be derived from either plant or animal sources. But Mother Nature has told our dogs that they should get most of their amino acids from animal sources. Why? Because:

  1. eating a lot of plant material irritates and can injure a carnivorous digestive tract; and
  2. animal protein is a higher quality source of protein.

What is meant by saying that animal protein has more quality (i.e. – “higher Biological Value”)? Well, lets say the food manufacturer learns of a pharmaceutical company that is going out of business and this pharmaceutical company has tons of the essential amino acid tryptophan. So the dog food company buys it and that is its source of protein in the diet. And let’s say they put enough of it in each bag that they can honestly say on the label: 30% protein. Sound good? Sure it does. The only problem is that your dog will soon be dead. It is not enough that the
food have a certain amount of protein; it has to have it in a proper balance of each amino acid to every other amino acid. That balance is reflected in proteins derived from animal sources; it is not reflected in proteins derived from plant sources. Yet 40% or more of the protein contained in the PREMIUM BRANDS of dog food (and don’t you get me started on the other feeds) is derived from plant sources. Now it is possible for the plant and animal source proteins to be balanced and result in a food with a high Biological Value (putting aside all concerns about all that plant matter which will irritate the dog’s gut) but how are you going to know if your feed manufacturer did this balancing act or just threw in “22% protein”?

Lastly we come to the bit players: vitamins and minerals. I say “bit players” not because they are unimportant but because just a little of them goes a long way. Vitamins do not provide either fuel energy nor structural components for the body. They act as catalysts, instigating essential chemical reactions in the body without actually becoming part of the reaction. Minerals have a less specific role, some acting as structural components (e.g. – calcium being used in bone development), some as catalysts, some as regulators. Like amino acids, minerals demand a balance amongst themselves and inadequate or excessive amounts of one will create a systemic imbalance of the whole array of minerals.

The proportions of these nutrients required by our dogs is not only different than the proportions required by us but, like us, also varies depending on the age and activity of the dog. Therefore the pet food industry is right about having different formulas for growth, maintenance, athletic performance, and seniors. (Whether they have actually formulated them right is another matter entirely.) It seems that puppies need considerably more protein, for example, than an adult dog. Their little bodies not only are doing all the reactions that adult bodies are, but also building the muscles, hormones, enzymes and immune systems that contain those reactions. An athletic dog, on the other hand, needs slightly more protein than a sedentary dog (not much more, contrary to popular myth) but far more energy (preferably fat and not carbohydrates). There are all kinds of tables I could reproduce that show the exact proportions, except for one thing … I don’t trust those tables. You see, those studies are based upon consumption of commercial dog foods with their high vegetable content, low fat content and questionable protein sources. I am not aware of any tables which are based on more objective sources, such as diets of feral dogs, wolves and coyotes.

Let’s look at the growth phase, for example. Research says that feeding puppies energy rich diets is undesirable. We are routinely told to underfeed our puppies, because to do otherwise leads to rapid growth. Rapid growth in turn is closely linked to undesirable decreases in bone density, higher body weight and eventually dysplasia. But it turns out that the subject of these studies was puppies whose exercise was restricted. Now, can you imagine a less natural condition than a group of puppies not allowed to run and tumble and fight and do all the things that burn calories and prevent puppies from getting fat? And what do you know: when these studies were recently reproduced EXCEPT the puppies were allowed to play and exercise at will, there was no such accelerated growth rate, no fat puppies and no bone disorder. So my query is this: how can we trust the existing numbers (X% protein, Y% fat and Z% carbohydrate) when they are based on studies of how to economically feed dogs on pre-existing commercial rations rather than studies on what canines naturally eat to achieve and maintain a healthy condition?

A nice little example is the long standing warning against feeding your dogs too much protein for fear of overworking the dog’s kidneys. You see, protein (amino acids) contains nitrogen at its core. Any excess nitrogen (“excess” meaning it is not needed for the building blocks but used instead as a mere energy source) has to be removed (the process is called deamination) by the kidneys and excreted as urea in the dog’s urine. Now ultimately this information is true. But our preconceptions, based upon our own omnivorous diets, of what level of protein consumption is ideal, has little applicability to a carnivorous canine. This information can only be obtained from studies of our canine friends. And such studies as have been done have focused on commercial diets which bear no relationship whatsoever to to what a dog eats when he has other choices, such as protein from animal sources. And, keeping in mind that it is the whole spectrum of animo acids that a dog needs and not merely a bulk quantity of “protein”, what proportion of protein is ideal will vary widely depending on the quality of the protein (with egg protein being the ultimate, various meats being good, and plant sources being less valuable no matter how much “protein” they supply).

Another kink in the question of “how much of each nutrient should I feed my dog” centres around puppies. New research on the growth phase has found that nutritional requirements differ by “type”. That is: toy breed puppies have different requirements than giant breed puppies. What is suitable for one type of puppy, then, is not necessarily suitable to another puppy which, at adult size, will be substantially larger than the first puppy.

Another problem facing us is that research has centred around processed foods rather than the foods that our dogs are naturally happy with. Who knows the effect of cooking on the quality of the nutrient to the dog? And what about the dyes (like Red Dye 40 which is banned for human use) often used in dog food to make us buy it because of its attractive appearance? Related to this is the manner in which nutrients are preserved. Most common are synthetic preservatives like:

  1. BHA and BHT: used to preserve fat and linked to liver and kidney failure, birth defects, allergies and various other afflictions;
  2. Ethoxyquin: also used to preserve fat (and the unrelated use as a rubber hardener) and linked to immune system disorders and cancer as well as a list of other disorders.

In reaction to public concern over such additives many lines have come up with the idea of using the natural preservatives of vitamins C and E. Unfortunately, what they haven’t told us is that it appears that the vitamins rapidly deteriorate the moment the food is exposed to air (i.e. – once the bag is opened).

As if all this wasn’t enough to give you a headache, consider some other dirty tricks allowed in the pet food industry:

  1. While the ingredients are supposed to be listed in descending order of quantity, some such as whole chicken are now apparently allowed to be listed on a fully hydrated basis. That is, chicken may be first on the list but that’s with all the water still in. When it gets to you all the water is out and that chicken may be more appropriately the fifth or sixth ingredient,
  2. Not first; some manufactures may list, for example, corn meal, kibbled corn and corn oil as ingredients 5, 6 and 10. But what are all three of them: corn. And when you lump them together like any normal person would do … ta da: ingredient #1 is in fact (though not on the label) CORN; there is no apparent regulation of “meat by products”, so some reputable manufacturers may be using quality by products like brains and eyes, while others are using feathers and manure.

I am afraid I have more questions than I do answers, but I hope that if nothing else my questions will make you look at your dog’s diet and wonder: is this the best I can do for my friend?

Dilated Cardiomyopathy

Dilated  cardiomyopathy (DCM)

is a disease of the heart muscle which causes the heart to enlarge and not function properly. The occurrence of DCM usually  increases with age and typically has an age of onset between 4 and 10 years. The cause is still unknown although many factors strongly suggest a genetic cause.

It usually affects both the left and right sides of the heart with either side being more severely affected. Typically both the lower chamber and the upper chamber enlarge and the lose their ability to contract and pump blood out to the body or the lungs. The consequence of this can be compared to a simple mechanical pump, which, if it fails, water backs up into the basement. Therefore, if the left heart fails, fluid backs up into the lungs and if the right heart fails, fluid backs up in the abdomen or in the space surrounding the lungs.

Long term prognosis varies considerably. Most dogs survive from weeks – 24 months of age upon diagnosis of DCM.

Treatment is aimed at improving the heart’s function and controlling the signs of congestive heart failure. Drugs such as Lanoxin, Digoxin and Digitalis are used to help the heart contract better. Diuretics such as Lasix (Furosemide) are used to help control and prevent accumulation of fluid in or around the lungs. Vasotec, Enacard, Zestril, Prinavil, and Lotensin are used as well to help the heart pump more effectively against the pressures of the arteries and veins. You and the dog will need to see your regular veterinarian in 7 to 10 days, in 4 weeks and then every three months to assure that kidney function is preserved. Drugs that control cardiac arrhythmias (electrical disturbances in the heart) are used as well.

It is important for you to monitor your dog’s overall attitude and outward signs so that if you notice any heavy/labored breathing, coughing, fainting spells, restlessness or profound lethargy, arrangements can be made to see your regular veterinarian quickly. Your observations the administration by you of the prescribed medications is what will help your dog the most. You know your pet the best.

Please feel free to check out the links supplied below:

 


 

Cardiomyopathy in the Doberman breed

by Judy Doniere, Toledobes, USA

When Peggy Adamson wrote of the 7 sires all dying of Cardiomyopathy she wrote of the popular dogs on the East coast that were being used and were in many many pedigrees. However, many people were importing dogs from Germany. Some went to the West coast and a great many went to the Midwest.

If we only had 7 sires to use, the breed would be so inbred that if they all had or died of Cardio, no Dobermans would have been around several generations later.

One of the bigger Kennels was Ponchartrain in Detroit. They did use a couple of the 7 sires but they also imported many dogs. In Chicago there were many kennels that imported dogs. Few used any of the 7 sires.

We only hear of the males that were imported but a great many bitches were also imported and few of them went back to the 7 sires.

We do have lots of dogs and bitches who live to 12 and some to 15. The larger the dog (breed) the shorter the life span. Toys live into the late teens time and again.

A Doberman of 12 is an old dog. Am Ch Toledobes Serenghetti aka Sera lived to be 12 but her 2 litter brothers died at 8 from cardio. One of her aunts lived to be 17. I´ve also had a few live to 15.

I think now that we have methods of freezing semen, we should do it on any of our 2 year old dogs. By then you should know how good they are. If they live beyond 10 use them, but you can still have Cardio on a 10 yr old although the chances are good that you might get lucky.

There are no more LINES of dogs in this country. In the early years everyone linebred or inbred. That´s partly why no dogs look alike due to their genes being spread wide so that you have no idea what you´re going to get.

A few kennels have had a couple of good dogs and then they linebred and inbred them until they started going backwards since there were no more genes bringing in good qualities and they had to go completely out so now their dogs look like everyone else´s.

The dog that I know that died of Cardio and produced it because he was so line bred was Prince Kuhio. Many of his get DID live a long time but many died early. Then people in Ohio started breeding everything to him and then kept doubling up on him, so eventually their dogs started dying early of DCM. I´m sure if you trace your pedigrees back far enough, you will find him back there. Not in ALL pedigrees but enough of them to be able to pinpoint where a lot of DCM came from and yet, he does trace back to a couple of the 7 sires at least through Uranus.

Diet And Your Pet

by Ms Dany Canino

Too Much Of A Good Thing Can Be Hazardous To Your Pet

Every pet owner has one main objective in mind for their pet; to give the pet the very best and, at the top of this list is, “food”.

Unfortunately, far too often pet owners tend to overdo in this area. They feel that the animal needs a lot of protein for energy, stamina, and general nutrition.

Protein does offer energy and stamina to the pet, but just how much energy does the pet need?

If this is an animal that is working on the farm alongside his owner for 6 hours a day, then that animal can utilize a good amount of protein in his system. If this animal is a working sled dog he will burn up any excess protein. If he is a working Police Dog he will utilize high amounts of protein through his work and the stress he´s under.

Most family pets spend their days lazing in the backyard or on a couch. An excessive amount of protein offered into their system lies dormant, in other words, un-utilized. Therefore, one of two things can occur: 1) the pet gets fat and/or starts scratching a lot therefore, causing the pup to irritate his skin. 2) When the animal eliminates this food, it might tend to smell like undigested food and the animal will start a nasty habit of eating his stool.

Animals eat for caloric intake. If you are feeding a good quality food that has been purchased from a reputable pet supply store, your dog won´t need as much food as he would if he were to be fed a commercial dry food purchased from the supermarket. Most supermarket dog food has a lot of sugar in it, so the dog is enticed to eat. It also tends to have a lot of salt in it, so the dog is thirsty after eating and drinks a lot of water. This gives him a sort of “false full”. However, as most owners have discovered, dogs fed this way tend to go to the bathroom very frequently. This means that they are left with very few nutrients in their system to keep them healthy and hardy. This dog always seems hungry so you feed him more and more. “Good food” satisfies the dog´s needs in smaller amounts.

Puppies need more caloric intake and protein than adult dogs do to help in proper growth. Pregnant females need more calories and protein because the puppies inside her are taking up a lot of her nutrition. However, in either case these animals should not need more than about 26% to 28% protein for short periods of time. Prolonged use of this diet could be detrimental.

At about 6-8 months of age the puppy can be changed over to adult food with a protein level of about 20%-24%. (Myself and other breeders have switched pups over to adult food as young as 5 months without any ill-effects whatsoever.) There´s no proven data to attest that pups need to be on puppy food past 5-6 months, nor is there any data to prove that the dog´s growth is inhibited by taking him off puppy food before 1 year of age. If your pet is on a good puppy or adult food you shouldn´t have to add any vitamins to this diet. The exception to that would be if this were a pup that was teething. The calcium he might need for other parts of his growing body would be used up for his new teeth to come in. Therefore, at this time you might want to add some good “oyster shell” calcium as a supplement. You can purchase this at any drugstore or health food store. (Oyster shell is one of the best sources of calcium available.)

One vitamin you should consider offering to your dog lifetime is Vitamin C. Dog´s do not produce this vitamin in their system so we need to offer it into his system. It´s a vitamin that helps to ward off infections, helps your dog to assimilate his food and, some breeders feel it aids healthy bone and joints. I prefer a natural vitamin C (ascorbic-acid) given in powder form, but you may also give it in pill form. On a large dog you can give as much as 1000 mg as it is a water-soluble vitamin and any excess will be eliminated through the dog´s urine. Ask your local pet supply dealer about this product. If your household is a smoking household this vitamin is absolutely necessary because secondhand smoke affects an animal just as it affects a human.

A dog should be switched over to senior food (or less active) at about 6 ½ years of age. This is usually when their activity level wanes. By doing this, your dog´s pancreas doesn´t have to work as hard to digest the protein intake.

Moist food is a fine additive to your dog´s food but this too, is usually given in excess to a dog. Moist foods should mainly be given for flavor. These foods are usually only 25% nutrition and 75% water. Therefore, moist food should never be used as a staple food. Large dogs should only be given about 2 tablespoons. Medium sized dogs should only be given about 1 tablespoon. Small dogs should only be given 1 teaspoon. “Just Think Of The Savings”! By feeding a good quality dry food that you can feed less of and, by adding only enough moist food to flavor, you´ll easily save as much as $10.00 a month. Remember, you don´t have to add moist food. However, if you prefer to feed just dry food, moisten the dry food with water before feeding so that you reduce the chances of torsion bloat in your dog.

There is a medical condition in dogs that has become all too common. That problem is “hypothyroid”. Having your Veterinarian run a simple blood test on your dog for a “T3 – T4”, easily discovers this malady. This will determine if your dog has this problem and will enable the Vet to prescribe medication to control this problem. Some symptoms of this problem are: Lethargy – Weight Gain – Hair Loss – Drastic Change In Disposition. If you dog has exhibited any of these symptoms you would be wise to have him tested. (Certain breeds are prone to hyperthyroidism. Ask your dog´s breeder or your Veterinarian if your dog´s breed is a candidate for this problem.)

You really need to learn to read the labels on the food your dog is going to ingest. The old adage is true that; “you are what you eat”. If you see words like; “ethoxoquin” (a chemical preservative) – “sodium” (salt) – “sucrose or dextrose” (sugar) or an excessive amount of “BHA” (another chemical), and if these products are shown to contain an excessive amount, you need to look for another food. The best rule of thumb to follow is that the first four ingredients are the most important things in determining what you´re feeding your pet.

So feed your dog a good nutritional diet (2 meals a day for life is best) and take him for walks, or give him some other type of exercise so that he can utilize the food you´re feeding him. He´ll be healthier and live longer.

NOTE: I always add a small can of vegetables to my dog´s food. I use carrots, green peas, green beans, and sometimes a little pumpkin. Dogs love this tasty addition to their food and it´s also quite healthy. For treats and snacks I give my dogs whole carrots instead of cookies and rawhide.

Diarrhea-causes & Remedies

written by Robert Hart, Vitality Science

Man´s best friend tends to have a strange palette. Rarely will a dog pass up eating a bug, or a funky morsel: the funkier, the better. Should you happen to catch them in the act, they will often gulp it down before you can extract it from their vice-like grip. No telling how many different kinds of bacteria such morsels are harboring.

However, their palette is not restricted to bugs and food morsels. Food wrappers, aluminum foil, tinsel seem to be enjoyed by dogs. One has to wonder if part of the driving force behind CDs and DVDs was eliminating cassette tape. This, too, shall pass.

Such indiscrete eating habits often lead to diarrhea. Many times, you only the effects of what they ate. And where do they throw up?

It seems like your most expensive throw rug is the flooring of choice- of course. But they look up at you with such sad eyes (do you think they practice it when you´re not home?) it´s hard to scold them.

Other possible causes:

Diet Change
Merely changing brands of dog food can cause a few days of vomission. Many commercial dog foods contain high levels of indigestible protein. But any change, even to raw, should be done gradually to avoid the problem.
Eating Too Much and Too Fast
There are a number of reasons why dogs eat too fast. Competition from other household dogs and dogs not fed frequently enough may vomit undigested food soon after eating. Smaller dogs should be fed less food, but more often. If they are part of a pack of dogs, feed them in a separate room from the bigger dogs.
Table Scraps
Most table food has more nutritional value than most commercial pet food. However, dogs should not be given spicy scraps or rich foods. Both go down fast, but come up just as fast.
Water

Municipal water in the US contains chlorine and possibly ammonia. They are necessary to make sure any bugs in our water are dead, but… you don´t want to drink them because they can kill the bacteria in the intestinal tract that is necessary to maintain balance. You must be mindful that water free of disinfectant can quickly breed bacteria, which can also cause diarrhea.

Cleaning Products

If you by concentrated cleaners and do not dilute them sufficiently, your dog can breath in toxic fumes. Also, strong cleaning agents can burn a dogs pads. They will lick their pads to stop the burning and ingest the chemicals which can also kill the bacteria in the GI tract, causing diarrhea.

Remedies

Vomiting can be curative in itself, in that it purges spoiled food and toxins from the stomach and intestine.

A large dose of an all-natural laxative will help move the material to move through the colon.
A natural remedy gaining popularity is to administer soil-based organisms (SBOs).
Monitor your dog´s demeanor and stools carefully for seventy-two hours to be sure the material passes and their energy improves.
If after giving them SBOs or an all-natural laxative, but do not see an improvement in demeanor and energy, make an appointment to see your vet.
Limit your dogs´ food to clear fluids and begin gathering stool and vomit samples. Time date them for your vet. You might want to store them in a cool place, like a cooler.
If your dog has strong digestive odors, it could have an intestinal obstruction.

Color Dilution Alopecia

written by R.M. (Bug ) Russell

The dilute colors (fawn and blue) do have coat and skin problems and there is a ton of available information on them. There is one major problem called CDA (see below) that is an alopecia (hair loss) directly related to the coat color. The granules of material which provide the color of the coat are located in the hair shaft…in blacks and most reds and fawns and blue who don’t have CDA the material is evenly spread through the hair shaft. In the CDA dogs it is found in clumps…the clumping evidently makes the hair shaft fragile at that point and prone to breaking. When this happens below the skin line in the follicle itself it can “kill” the follicle so no new hair is produced. That’s the abbreviated version of the major problem.

Dobes in general are rather prone to staph infections…their immune systems are rather slow to mature…in a black and most reds this may be a problem in puppies and may result in temporary hair loss but in blues and fawns it can be a disaster coupled with CDA.

The rarity of seeing these colors in the ring is due in part to the difficulties in keeping them in good coat. Barbara Russell, who sort of specialized in dilutes, specifically blues, had blue Dobermans that generally had good coats she states that was that even with good blue coats she had problems and keeping blues in show condition as far as coats went was a full time job.

There are definitely some judges who aren’t fond of the dilutes and a few that wouldn’t put one up under any circumstances. In the ’60s it was harder for reds to win than blacks…I think that is about where the dilute situation in the ring is now…it’s harder to win with a dilute but not impossible if the dog is a good specimen of a Doberman.

The dilute colors are produced much less often than reds or blacks. Genetic statistics say that fawns comprise about 6% of all Dobermans born and blues somewhere between 12 & 15%…all the rest are black or red but I can’t remember what the stats are for them).

Thin coats on the dilute colors are not usually due to allergies and the dilutes don’t seem to have any more skin problems, with the exception of CDA, than blacks or reds do.

If the coat looks good from 2 or 3 feet away it probably is good but most puppies in dilute colors have decent enough coats the thinning due to CDA takes place over time so a dog who had a decent coat at 10 months might well be bald at 5 years.

The dermatology texts say that over 90% of all blues will at least have thinning hair and many will thin to the point of being bald over most of the body. Fawns seem to have a better chance of retaining their coats with about 75% of the fawns having extensive hair loss due to CDA.

The literature also says that the darker the coat color in a dilute dog (steel blue in blues and carmel in fawns) the better the chance they will retain most to all of their coats. This seems to be the case in the blues and fawns I’ve known over the years.

There are a few dilute dogs whose coats are fine, who don’t lose hair, don’t have CDA and never go bald but they are few and far between.


COLOR DILUTION ALOPECIA

By Teri Dickinson, DVM

Alopecia (hair loss) related to dilute coat color is a recognized condition in dogs. The currently accepted medical terminology for this condition is Color Dilution Alopecia (CDA). The condition may affect any dilutely pigmented dog, regardless of coat color. This condition was previously known as Blue Balding Syndrome, Blue Doberman Syndrome, Color Mutant Alopecia, Congenital Alopecia, etc. The term Color Mutant Alopecia arose because dilutes were at one time mutations from the deep pigment occurring in wild canines. Dilutes are now a regularly occurring form of pigmentation in many breeds and have been for hundreds of years. The term mutation is therefore not applicable to dilute individuals. References to Doberman Pinschers or blue hair coats arose because the condition is common in blue individuals of this breed, but it is not limited to either blue dogs or Dobermans. The term congenital means present at birth, but CDA affected dogs are born with normal hair coats.

The dilute (also known as Maltese) gene also appears in both mice and cats, and interestingly enough, is not associated with any abnormal coat conditions in those species.(1) Color Dilution Alopecia (CDA) has been recognized in dilute individuals of many breeds of dogs including Chow Chows, Dachshunds, Doberman Pinschers, Great Danes, Irish Setters, Italian Greyhounds, Standard Poodles, Salukis, Whippets, and Yorkshire Terriers.((2),(3),(4),(5)) Dilute individuals carry a recessive genotype of dd and are characterized by blue, bluish-grey, lavender or flesh-colored noses, lips and eye rims. The coat colors may include blue, fawn, blue-fawn, bronze, taupe or some variation of these. These dogs are usually easily distinguished from their deeply (non-dilute) pigmented counterparts. Deeply pigmented individuals carry a dominant genotype of Dd or DD and have black or liver noses, lips and eye rims. Coat colors may include black, red, red-fawn, liver or variations thereof.

CDA is characterized by loss of hair from dilutely pigmented areas. Coats are normal at birth, and onset of hair loss usually begins between six months and three years of age. Hair loss usually begins along the dorsal midline (middle of the back) and often spares the head, tail and limbs. The pattern seems to vary from breed to breed. It has been suggested(6) that darker colored (steel blue) individuals are less likely to be affected, may be less severely affected or may start to lose hair later in life than lighter colored dogs. This suggests that the severity of the disease may be related to the amount of dilution present. Deeply pigmented or white areas of coat are unaffected. In blue dogs with tan points (Yorkies and Dobermans) the tan areas retain a normal appearance. In piebald (white spotted) individuals, the white areas are unaffected by the hair loss. The hair loss may be total or partial and any remaining hairs are usually sparse, rough and easily broken or removed. The skin in the affected areas is usually scaly and may occasionally develop bacterial infections. Pruritus (itching) is usually absent, unless a bacterial infection has set in.

Diagnosis of CDA requires first ruling out other causes of hair loss. Diagnostic tests should include fungal cultures, skin scrapings to check for parasitic mites, etc. CDA often closely resembles endocrine (hormone related) hair loss and the dog should be carefully examined for any other abnormalities, and tested for normal thyroid function. Presence of dilute pigment and a characteristic course of disease also aid in making the diagnosis. Microscopic examination of hairs and\or skin biopsies can be used to confirm the diagnosis.

There is no cure for CDA. Treatment is limited to controlling the scaliness and any associated pruritus with various shampoos or topical treatments.

The cause of CDA is not clearly understood. Microscopic examination of hairs of dilute individuals reveals that the pigment (melanin) forms large granules (macromelanosomes) which are rarely found in deeply pigmented hairs. In dilute individuals with normal appearing coats, these macromelanosomes are not grouped or clumped and cause no distortion of the cuticle (outer covering) of the hair. Dogs with CDA have many large groups or clumps of macromelanosomes which tend to distort the cuticle of the hair. It is hypothesized that this distortion of the cuticle causes the hairs to break easily, resulting in the short stubby hairs commonly found in affected individuals. (See Drawing). It is further hypothesized that the rupture of the hair releases by-products of pigment formation, which are toxic to the hair follicles. Re-growth of broken hairs is reduced because of damage to the follicles caused by
th
ese toxins.

Why in some dilute dogs the macromelanosomes are clumped and in others they are not, is an interesting question at this time. The relationship between dilute pigment and hair loss is clear, but why are some dilute individuals unaffected? Weimeraners as a breed are dd, all individuals are dilute, yet the disease is unreported in this breed. In Dobermans, the dilute individuals comprise only 8-9% of the breed, yet 50-80%6 of the dilute dogs have CDA. In Italian Greyhounds, many individuals are dilutes, yet the IGCA health survey reported only 71 affected individuals among the approximately 2200 dogs included in the survey.(7) If half the dogs included in the survey were dilutes, the incidence of CDA in IG’s would be around 7% of the dilute population, as opposed to the 50-80% affected dilute Dobermans.

A third allele (dl) which is associated with CDA has been proposed.6 While this is a long way from being proven, it could help explain why some dilute animals are unaffected. Dogs with a genotype dd would be normal coated dilutes, ddl would be intermediates (mildly affected?) and dldl would be CDA affected. A genotype of Ddl should represent deeply pigmented dogs which were carriers of CDA.

Colloidal Silver Rediscovered

Prior to 1938,  Colloidal Silver was commonly used to combat up to 650 different microorganisms associated with diseases.

Colloidal Silver appears to be a very powerful, natural antibiotic and preventative against infections.  It acts as a catalyst disabling the enzyme that one-celled bacteria, viruses and fungi need for their oxygen metabolism by suffocating the microorganism.  The pathogen or micro-organism dies within minutes and is flushed out of the body through the immune, lymphatic and elimination systems.

Pharmaceutical antibiotics destroy beneficial enzymes such as those in the intestine which are the natural or good bacteria, whereas colloidal silver leaves all tissue cell enzymes intact.

Colloidal silver has a long medical history that is well documented with a broad antibacterial, antifungal and immunosuppressive properties.  It is also effective on many parasite eggs as well because the parasites usually have a one cell egg stage in their reproductive cycle.      

Modern  antibiotics kill perhaps half a dozen different disease organisms, but silver kills about 650 thereby not allowing resistant strains to develop.  As well,  silver is virtually non-toxic. Most of our `super-bugs´ or `super-germs´ today are caused by the microorganisms becoming resistant to modern antibiotics because of overuse of antibiotics.  It is believed that single celled germs or micro-organisms can´t mutate into silver-resistant forms.

Antibiotics absorb  so slowly that they travel to your intestines killing many of your normal, healthy, friendly bacteria, whereas colloidal silver is absorbed into the blood before it reaches the intestines.

Ionic Colloidal silver is the premier immune system booster, plus it is a proven immunosuppressive and anti-inflammatory NATURALLY.

Below is a partial list of some different uses for Colloidal Silver for both humans and animals and plants :
acne AIDS  allergies
arthritis  athlete´s foot UTI’s
boils burns cancer
candida cholera colitis
conjunctivitis cystitis dermatitis
diabetes diarrhea eczema
fibrositis       gastritis hay fever
herpes impetigo indigestion
leprosy leukemia lupus
lyme disease malaria meningitis
parasitic infections : viral, fungal, and bacterial pneumonia pleurisy prostate enlargement
psoriasis pruritus ani (itchy anus) ringworm
sinus infections septic conditions of the eyes, ears, mouth and throat septicemia
shingles skin cancer staphylococcus 
streptococcus  stomach flu thyroid
TB toxemia tonsillitis
warts   yeast infections  whooping cough   
stomach ulcers canine parvovirus fungal and viral attacks on plants
soothes skin conditions  cleans wounds cleans eyes and ears
maintains a healthy digestive system juvenile pyoderma bug bites 
apply directly to cuts, open sores or on a bandage for warts spray pet bedding & let dry
dab on cuts, grazes, rashes, sunburns and razor nicks mix a little into the dog´s drinking water, birdbaths and cut flower vases to prevent water from forming algae or growing bacteria helps reduce tooth decay, mouth sores and bad breath by eliminating bacteria deep in the back of the throat and on back of tongue
add to human and pet shampoos for disinfectant benefits spray onto burns for rapid healing without scarring use to sterilize toothbrushes or washing brushes
spray on rubbish to prevent decay odours use on kitchen cloths and sponges to wipe down countertops and cutting boards dab onto acne 
add to bath water, gargle and nasal spray spray in shoes, between toes, on skin to give relief to itching, athletes´ foot, fungi aids recovery from colds, flu, pneumonia, respiratory infections and viruses
spray plant foliage to help prevent plant diseases like mold and rot wipe around toilet seats, bowls, tile floors, sinks, door knobs kills persistent odors
excellent as underarm deodorant because most underarm odor is caused by bacteria breaking down substances released by sweat glands.    
     

High concentrations of silver apparently do not kill germs more effectively than the safe range of 3-5 ppm (parts per million).  If micro-organisms are killed off too quickly the body´s five elimination systems or channels (liver, kidneys, skin, lungs, and bowels) may be temporarily overloaded, causing flu-like symptoms such as headaches, extreme fatigue, dizziness, nausea, or aching muscles.  If this happens, reduce the amount of colloidal silver a little and increase the amount of distilled water intake. Regular bowel movements are important to relieve the discomforts of detoxification.  Reduce sugar and saturated fats from your diet.     

For pets :  1 – 2 teaspoons daily either directly from a dropper into the mouth or in drinking water, or both.  It can be used directly on the skin to clean cuts, soothe eye or ear infections.

For humans :  1 – 4 teaspoons daily in considered a nutritional amount and is safe for extended periods of time whereas higher doses are considered to be `therapeutic amounts´  and should be used only occasionally.  It should be used in proportion to body weight.  There has never been a drug interaction reported between colloidal silver and other medications. 

When the possibility of germ exposure is higher, colloidal silver can be taken by mouth (orally) daily or applied topically (on the skin) when there is a skin problem.

Colloidal silver is tasteless and won´t sting even a baby´s eyes and won´t upset the stomach.  It promotes rapid healing with less scar tissue, even with severe burns. 

Digestion has been reported as far better.

The ideal color of colloidal silver should be a golden yellow.  

Chronic Active Hepatitis #2

Written by Dr. Alice Crook and taken from the Canine Inherited Disorders Database with permission

related terms: chronic liver disease, copper-associated hepatitis/copper toxicosis in Bedlington Terriers, chronic active hepatitis, Doberman hepatopathy, chronic inflammatory hepatic disease

What is chronic hepatitis?

Chronic hepatitis is a catch-all diagnosis for a number of diseases where there is inflammation and death of liver tissue. Affected dogs develop a slowly progressive liver disease. There are many possible causes including viruses, bacterial infection, and some drugs. A familial predisposition to the development of chronic hepatitis exists in some breeds, although the cause is unclear. These breeds are listed below.

In Bedlington Terriers and West Highland White Terriers, an inherited defect in the metabolism of copper is known to cause chronic hepatitis (copper-associated hepatitis/copper toxicosis in Bedlington terriers). It is estimated that 25% of Bedlington Terriers have the disorder, and 50% are carriers (i.e.. they will not become ill, but can transmit the disorder to their offspring). Copper accumulation occurs in some other breeds as well, but whether the excess in copper is the cause or the result of liver disease is unknown.

How is chronic hepatitis inherited?

Inheritance in the Bedlington Terrier is autosomal recessive. The  mode of inheritance is unknown in other breeds.

What breeds are affected by chronic hepatitis?

Doberman Pinscher (predominantly female dogs affected), American and English Cocker Spaniel, Skye Terrier, Labrador Retriever.

Copper-associated hepatitis – Bedlington Terrier and West Highland White Terrier 

For many breeds and many disorders, the studies to determine the mode of inheritance or the frequency in the breed have not been carried out, or are inconclusive. We have listed breeds for which there is a consensus among those investigating in this field and among veterinary practitioners, that the condition is significant in this breed.

What does chronic hepatitis mean to your dog & you?

The liver has a large reserve capacity, meaning there must be damage to a significant portion before you will see any signs of illness in your dog. The first signs of liver disease are generally vague and non-specific, and include loss of appetite, vomiting, weight loss, depression, lethargy, and/or increased drinking and urination. As the disease becomes advanced, there will be signs more specific to liver failure such as jaundice (you may see yellowing of your dog’s eyes for example), coagulation problems, fluid accumulation (ascites), extreme weight loss, and neurologic abnormalities (hepatic encephalopathy) such as head pressing or behavioral changes, due to the build-up of toxins normally metabolized by the liver.

In affected Bedlington Terriers, there is progressive accumulation of copper in the liver over time. Associated liver disease may manifest itself in 3 ways.

1) Young dogs (less than 6 years of age) may suddenly develop signs of liver failure, including vomiting, depression and lethargy. Most will die within a few days despite intensive therapy. Others will recover over a few weeks, and thereafter experience occasional milder bouts that may be associated with stressful events such as showing or traveling.

2) In middle-aged to older dogs, similar but less severe signs develop insidiously and the disease has a more chronic course. Gradual weight loss and deterioration in condition are common initially, while specific signs of liver disease (as listed above) develop in the advanced stages of the disease.

3) Young as yet clinically unaffected dogs may be found to have elevated liver copper and biochemical/pathological changes associated with liver disease. These are the dogs in whom treatment will be most effective.

West Highland white terriers are also affected by a disorder of copper accumulation, but the magnitude of increase is less. Diagnosis, signs of illness, and principles of treatment are similar to the Bedlington Terrier.

How is chronic hepatitis diagnosed?

Before liver disease reaches an advanced stage, the clinical signs are generally vague and non-specific. Your veterinarian will do some routine diagnostic blood tests which will show elevations in liver enzymes. This is investigated further with specific liver function tests. Once the problem has been pin-pointed to the liver, your veterinarian will take a liver biopsy which will be sent to a veterinary pathologist. This is necessary to differentiate chronic hepatitis from other causes of liver disease (such as liver cancer or an infection), and to determine the severity and extent of the problem.

For the veterinarian: Elevated ALT is the most consistent biochemical abnormality. With acute onset hepatic necrosis there may be hepatomegaly.  With chronic advanced disease, the liver is small and not palpable due to fibrosis and cirrhosis.

Liver biopsy is required for definitive diagnosis of chronic hepatitis, to characterize the extent of fibrosis and progression to cirrhosis (which is by definition irreversible and indicative of a poor long-term prognosis), and also to quantify hepatic copper levels. The key histopathologic feature is moderate to severe inflammation. The role of hepatic copper accumulation is controversial. Copper is commonly elevated, but this is believed by most to be secondary to the chronic inflammation, in contrast to the primary copper accumulation that causes copper-associated hepatitis in Bedlington and West Highland white terriers.

Copper-associated hepatitis should be suspected in any Bedlington terrier with physical or biochemical evidence of liver disease, and biochemical screening should be done in any Bedlington terrier with a vague illness. Asymptomatic affected dogs can be detected through biochemical screening (2/3 will have elevated ALT), liver biopsy, or a radioisotope study using copper-64. Affected dogs will have reduced stool radioactivity compared with normal dogs, due to reduced biliary excretion of copper. A genetic marker has been identified for copper toxicosis in Bedlington Terriers that will detect affected and carrier dogs at any age – see www.vetgen.com

How is chronic hepatitis treated?

Unfortunately, liver damage is often advanced by the time the disease is recognized. Depending on the stage of your dog’s illness when it is diagnosed, treatment may involve intravenous fluids, antibiotics, corticosteroids, dietary management, and possibly medication to reduce copper levels in the liver. Your veterinarian will discuss with you the prognosis for your dog.

In Bedlington Terriers, there are a few different drugs to control copper accumulation in the liver. These must be given for the life of the dog to prevent further build-up of copper. If the disorder is detected early, when there are no or few clinical signs, these drugs will allow many dogs to live out a normal life.

For the veterinarian: In Bedlington Terriers, D-penicillamine (copper chelator which promotes urinary copper excretion), trientine hydrochloride (an alternative copper chelator) or zinc therapy (decreases intestinal copper absorption) may be used to gradually reduce hepatic copper content and prevent further accumulation. Treatment is similar in the West Highland White Terrier. Because copper accumulation is not continuous throughout life in the Westie, mature dogs may not require chelation therapy, depending upon hepatic copper levels.

Unfortunately, hepatic liver damage is generally advanced by the time a liver biopsy is taken and the disease is recognized, leading to a poor prognosis. Improvement may occur with prednisone therapy. The usefulness of copper chelation therapy in breeds other than the Bedlington and West Highland White Terrier remains unclear.

Breeding advice

A genetic marker has been identified for copper toxicosis in Bedlington Terriers that will detect affected and carrier dogs at any age – (see www.vetgen.com Because of the prevalence of this serious disorder in the Bedlington Terrier, all dogs to be used for breeding should be tested. The results may be registered with the Orthopedic Foundation for Animals (see www.offa.org) Testing may also be done by liver biopsy but dogs must be older than 1 year of age so that sufficient copper accumulation will have occurred to be detectable.

There is no similar testing available for other breeds. Affected dogs should not be bred, and breeding of their parents should be avoided as well.

FOR MORE INFORMATION ABOUT THIS DISORDER, PLEASE SEE YOUR VETERINARIAN.

Resources

Johnson, SE.1995. Diseases of the liver. In S.J. Ettinger and E.C. Feldman (eds.) Textbook of Veterinary Internal Medicine, pp. 1313-1357. W.B. Saunders Co., Toronto.

Leveille-Webster, C.R. and Center, S.A. 1995. Chronic hepatitis: therapeutic considerations. In J.D. Bonaguara and R.W. Kirk (eds.) Kirk’s Current Veterinary Therapy XII Small Animal Practice. pp. 749-756. W.B. Saunders Co., Toronto.

Twedt, D.C. 1997. Treatment of chronic hepatitis. ACVIM- Proceedings of the 15th Annual Vet. Med. Forum, pp.234-236.

Leveille-Webster, C.R. 1996. Medical management of inflammatory hepatic disease in dogs. ACVIM-Proceedings of the 14th Annual Vet. Med. Forum, pp. 43-44.

Orthopedic Foundation for Animals, Registry for Copper Toxicosis in Bedlington Terriers, www.offa.org